Why People Got IgA Nephropathy and the Treatment

Why People Got IgA Nephropathy and the Treatment
IgA Nephropathy is a type of kidney disease. Now More and more people have get it.IgA nephropathy also known as berger's disease, a look at what it is, causes, incidence and risk factors. And it is a kidney disorder that occurs when IgA-a protein that helps the body fight infections-settles in the kidneys.
IgA Nephropathy is a kind of immune disease with mucosal damage. The reason why people got the disease is that there was a time when you have loss of immunity and the germs and viruses would invade the body leading to infection in upper tract or gastrointestinal tract. This will make the IgA of abnormal structure have antigenicity and stimulate the body to produce corresponding antibodies thus forming complex of antigen and antibodies. These complex are highly compatible to the binding protein and acceptors of glomerular mesangial cells and the combination of the two will induce the proliferation, multiplication and activation of the mesangial cells and secrete inflammatory factors and activated compliments, which will lead the renal ischemia and anoxia thus causing inflammatory damage to the glomerular mesangial cells.
It is not a fear of getting IgA Nephropathy. As long as we find corresponding treatment methods can we get rid of the disease. Line of Treatment for IgA Nephropathy
Using adrenocortical hormone(prednisone) on alternate days is helpful for patients with urine protein 1g/d. Prednisone can also reduce proteinuria for patients with minimal change disease of IgA deposition. Combined use of cyclophosphamide, persantin and warfarin can reduce the urine protein, but it has no effect on Glomerular Filtration Rate(GFR).
The immunosuppressive agents in western medicine have good effect in anti inflammation and restraining abnormal immune reaction. However, the pathological process of the kidneys is complex. Inflammation is only one circle of it. Only anti inflammation cannot solve the problem completely. What’s more, restraining the immune reaction can only stop the production of more IgA or other deposition, but has no effect on the deposited immune complex. Long time use of immunosuppressive agents has many serious side effects.
But what are Micro-chinese Medicine? Treatment for IgA Nephropathy With Blood Urine
The expert group of Shijiazhuang Kidney Disease Hospital has put forward ‘Each disease has its medicine and each medicine has its effect’ in the symposium of Chinese medical treatment of Nephropathy and Uraemia which was organized by Chinese Medical Society. And has put forward the authority idea and method of treating kidney disease-- the Micro-Chinese Medicine Osmotherapy.
Its four remarkable characteristics of new treatment method, quick effect, lasting efficacy as well as low cost created innumerable impressive medical miracles which raised unprecedented sensation in the domestic medical field and caused high attention of the colleagues at home and abroad as well as the innumerable patients.
The Micro-Chinese Medicine Osmotherapy has remarkable characteristics which are as the followings:
1.The efficacy of the medicine is enhanced.
2.Having high pertinence of treatment.
3.The biological active of the medicine is strengthened.
4. The repair of the kidney is strengthened.
Shijiazhuang Kidney Disease Hospital use different methods to treat IgA Nephropathy. The traditional treatment is only aimed at removing the hematuria, but our treatment is aimed at the root cause of hematuria. That is focusing on the repair of glomerular basement membrane. It is combined therapy of both Chinese medicine and western medicine with Chinese medicine as the core. We use Chinese medicine to expand the blood vessels, anti inflammation, anti coagulation and degrade harmful materials. The treatment of Chinese medicine adopts new combination to improve the effect of Chinese medicine and western medicine and developed hospital preparations. Among which the most advanced treatment is Micro-Chinese Medicine Osmotherapy for blocking renal fibrosis.